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Investigators are particularly interested in determining the mechanisms by which effects observed in murine models relate to broader mammalian metabolic regulation, and whether different experimental conditions (young vs aged models, healthy vs metabolically compromised states) show differential responses to NNMT inhibition

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Reis AM, Kabke GB, Fruchtenicht AV, Barreiro TD, Moreira LF

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Metabolicepigenetic disruptions (such as aberrant tricarboxylic acid levels, histone acetylation, DNA methylation or RNA methylation) impair fetoplacental development, triggering recurrent miscarriage, pre-eclampsia or fetal growth restriction

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